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货号	产品名称	文献标题	IF	杂志名称
KTB1850	CheKine™线粒体呼吸链复合体Ⅰ活性检测试剂盒（微量法）	Selective degradation of tRNASer(AGY) is the primary driver for mitochondrial seryl-tRNA synthetase-related disease	19	Nucleic Acids Research
KTB1850	CheKine™线粒体呼吸链复合体Ⅰ活性检测试剂盒（微量法）	Bile acid metabolomics identifies chenodeoxycholic acid as a therapeutic agent for pancreatic necrosis	14	Cell Reports Medicine
KTB1850	CheKine™线粒体呼吸链复合体Ⅰ活性检测试剂盒（微量法）	An Fgr kinase inhibitor attenuates sepsis-associated encephalopathy by ameliorating mitochondrial dysfunction, oxidative stress, and neuroinflammation via the SIRT1/PGC-1α signaling pathway	7	Journal of Translational Medicine
KTB1850	CheKine™线粒体呼吸链复合体Ⅰ活性检测试剂盒（微量法）	Ribosome-targeting antibiotic control NLRP3-mediated inflammation by inhibiting mitochondrial DNA synthesis	7	Free Radical Biology and Medicine
KTB1850	CheKine™线粒体呼吸链复合体Ⅰ活性检测试剂盒（微量法）	An Fgr kinase inhibitor attenuates sepsis-associated encephalopathy by ameliorating mitochondrial dysfunction, oxidative stress, and neuroinflammation via the SIRT1/PGC-1α signaling pathway	3	Journal of Translational Medicine
KTB1860	CheKine™线粒体呼吸链复合体Ⅱ活性检测试剂盒（微量法）	An Fgr kinase inhibitor attenuates sepsis-associated encephalopathy by ameliorating mitochondrial dysfunction, oxidative stress, and neuroinflammation via the SIRT1/PGC-1α signaling pathway	4	Journal of Translational Medicine
KTB1860	CheKine™线粒体呼吸链复合体Ⅱ活性检测试剂盒（微量法）	An Fgr kinase inhibitor attenuates sepsis-associated encephalopathy by ameliorating mitochondrial dysfunction, oxidative stress, and neuroinflammation via the SIRT1/PGC-1α signaling pathway	7	Journal of Translational Medicine
KTB1860	CheKine™线粒体呼吸链复合体Ⅱ活性检测试剂盒（微量法）	Mitochondrial dysfunction in metabolic disorders induced by per-and polyfluoroalkyl substance mixtures in zebrafish larvae	3	Environment International
KTB1860	CheKine™线粒体呼吸链复合体Ⅱ活性检测试剂盒（微量法）	Mitochondrial dysfunction in metabolic disorders induced by per-and polyfluoroalkyl substance mixtures in zebrafish larvae	12	Environment International
KTB1870	CheKine™线粒体呼吸链复合体Ⅲ活性检测试剂盒（微量法）	An Fgr kinase inhibitor attenuates sepsis-associated encephalopathy by ameliorating mitochondrial dysfunction, oxidative stress, and neuroinflammation via the SIRT1/PGC-1α signaling pathway	7	Journal of Translational Medicine

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